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Tumor-suppressor genes can be difficult to target with drugs because the mutant genes lack function.

PARP inhibitors that lead to double-strand breaks in cells with defective DNA repair mechanisms may increase apoptosis to treat some types of cancer.
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Within a single tumor, all the cells may not have the same gene mutations and thus may not be affected by the same drugs.
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Most anticancer drugs inhibit tumor-suppressor genes that have gain-of-function mutations.


PARP inhibitors that lead to double-strand breaks in cells are an effective treatment for cells with defective apoptotic pathways.

All tumors of the same tissue-type should be treated with the same drug, regardless of the individual mutations revealed by cancer-genome sequencing.