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given that caffeine blocks the activity of camp phosphodiesterase, propose a mechanism by which caffeine ingestion leads to heightened alertness and sleeplessness. given that caffeine blocks the activity of phosphodiesterase, propose a mechanism by which caffeine ingestion leads to heightened alertness and sleeplessness. camp phosphodiesterase catalyzes the conversion of atp to camp . caffeine can prolong or enhance the effects of epinephrine by reversing the direction of the reaction performed by camp phosphodiesterase. camp phosphodiesterase breaks a phosphodiester bond in a camp molecule. caffeine can prolong or enhance the effects of epinephrine by inhibition of camp degradation by camp phosphodiesterase. caffeine blocks the activity of camp phosphodiesterase but initiates a signal transduction pathway that leads to heightened alertness and sleeplessness (effects similar to epinephrine action) by means of other messenger molecules. camp phosphodiesterase metabolizes camp into atp . caffeine can prolong or enhance the effects of epinephrine by inhibition of camp recycling by camp phosphodiesterase

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A phosphodiester link in a cAMP molecule is broken by the enzyme cAMP phosphodiesterase. As cAMP phosphodiesterase is inhibited, caffeine can prolong or amplify the effects of epinephrine.

What occurs when phosphodiesterase is inhibited?

Phosphodiesterase is unable to degrade cAMP and cGMP when it is inhibited. As a result, their concentrations inside the cell rise, which in turn causes calcium concentrations to drop.

What impact does a phosphodiesterase type 5 inhibitor have?

The most often reported adverse medication responses include headache, flushing, stuffy nose, nasopharyngitis, and dyspepsia. PDE5 inhibitors have been associated with rare but serious reports of prolonged erections lasting more than 4 hours and priapism (painful erections lasting longer than 6 hours).

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