Genetic mutations that result in myosin protein malformations directly affect the function of smooth muscle, and cardiac and skeletal tissues.
Small genetic mutations in the myosin gene can change the shape of the protein. This can change how it interacts with actin, changing the molecular machinery that makes muscles contract. In some cases, a gene error can cause the heart muscle wall to thicken, a condition called hypertrophic cardiomyopathy.
Myosin is a thick filament-forming protein whose job is to pull on actin when muscle contraction occurs. In muscle tissue, there is a protein myosin which plays an important role in muscle contraction. Smooth muscle, cardiac muscle, and skeletal muscle have the protein myosin for contraction. If a mutation occurs in the myosin-forming protein, the function of smooth muscle, cardiac muscle, and skeletal muscle are also disrupted.
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